CORTEZ格蘭氏軍團(tuán)菌核酸檢測(cè)試劑盒

廣州健侖生物科技有限公司

廣州健侖長(zhǎng)期供應(yīng)：軍團(tuán)菌、諾如病毒、流感病毒等傳染病系列的快速檢測(cè)試劑盒。

軍團(tuán)菌的檢測(cè)試劑盒包括：軍團(tuán)菌尿液抗原檢測(cè)試劑盒、軍團(tuán)菌抗體快速檢測(cè)卡（膠體金法）、軍團(tuán)菌抗原快速檢測(cè)卡（膠體金法）、軍團(tuán)菌水樣檢測(cè)試劑盒、軍團(tuán)菌乳膠凝集試劑盒（軍團(tuán)菌診斷血清）、嗜肺軍團(tuán)菌核酸熒光PCR檢測(cè)試劑盒。

我司還提供其它進(jìn)口或國(guó)產(chǎn)試劑盒：包括傳染病系列、免疫組化系列、診斷血清等產(chǎn)品。

歡迎咨詢

歡迎咨詢2042552662

CORTEZ格蘭氏軍團(tuán)菌核酸檢測(cè)試劑盒

實(shí)驗(yàn)步驟

1） 將所有的材料和樣品都平衡至室溫（2-30℃）

2） 將所有的檢測(cè)卡從密封的試劑袋中取出。

3） 將樣品點(diǎn)滴器垂直置于樣品孔上方，向樣品孔中加入3滴樣品（120-150ul）。

4） 10分鐘內(nèi)讀取結(jié)果，強(qiáng)陽(yáng)性樣品可能會(huì)早點(diǎn)出現(xiàn)結(jié)果。

注意：10分鐘后讀取的實(shí)驗(yàn)結(jié)果可能會(huì)不準(zhǔn)確。

結(jié)果說明

陽(yáng)性結(jié)果：檢測(cè)線區(qū)域出現(xiàn)明顯的粉色條帶，另外質(zhì)控線區(qū)域出現(xiàn)粉色條帶。

陰性結(jié)果：檢測(cè)線區(qū)域不顯色，質(zhì)控線區(qū)域出現(xiàn)明顯的粉色條帶。

無效結(jié)果：靠近檢測(cè)線的質(zhì)控線在加樣品后15分鐘內(nèi)不可見的話，則實(shí)驗(yàn)結(jié)果無效。

7、產(chǎn)品特點(diǎn)
★操作簡(jiǎn)便，無需其它儀器和試劑，易于在各級(jí)醫(yī)院推廣；
★反應(yīng)迅速，5分鐘內(nèi)即可得到結(jié)果；
★結(jié)果清晰，易于判定；
★敏感度高，特異性強(qiáng)。

想了解更多的產(chǎn)品及服務(wù)請(qǐng)掃描下方二維碼：

【公司名稱】 廣州健侖生物科技有限公司

【市 場(chǎng) 部】    楊永漢

【】 

【騰訊Q Q】 2042552662

【公司地址】 廣州清華科技園創(chuàng)新基地番禺石樓鎮(zhèn)創(chuàng)啟路63號(hào)二期2幢101-103室

2、顱內(nèi)占位性病變（intracranialspaceoccupyinglesion）:在顱 腦損傷顱內(nèi)壓增細(xì)菌及腦腫瘤晚期，一般皆表示已發(fā)生小腦幕切跡疝 。表現(xiàn)為病側(cè)瞳孔擴(kuò)大及光反應(yīng)消失，對(duì)側(cè)肢體可出現(xiàn)癱瘓，繼之對(duì) 側(cè)瞳孔也出現(xiàn)擴(kuò)大，同時(shí)伴有意識(shí)障礙。根據(jù)病史及頭顱CT檢查多能 明確診斷。3、海綿竇血栓形成及竇內(nèi)動(dòng)脈瘤:可表現(xiàn)為海綿竇綜合征，除了動(dòng)眼 神經(jīng)癱瘓外，還有三叉神經(jīng)*支損害，眶內(nèi)軟組織，上下眼瞼、球 結(jié)膜、額部頭皮及鼻根部充血水腫，眼球突出或視乳頭水腫，炎癥所 致者常伴有全身感染癥狀，結(jié)合眶部X線片及腰椎穿刺及血常規(guī)檢查可 明確診斷。4、眶上裂綜合征與眶尖綜合征:前者具有動(dòng)眼、滑車、外展神經(jīng)與三 叉神經(jīng)*支功能障礙，后者除此3對(duì)顱神經(jīng)損害外，常伴有視力障礙 ，結(jié)合眶部視神經(jīng)孔X線片，血液化驗(yàn)、眶部CT等多能明確診斷。5、腦膜炎:腦膜炎引起的動(dòng)眼神經(jīng)損害多為雙側(cè)性，且多與滑車、外 展神經(jīng)同時(shí)受累。腦脊液檢查細(xì)胞數(shù)、蛋白定量增細(xì)菌。關(guān)鍵診斷細(xì)菌素上瞼下垂和斜視 (常見)： 根據(jù)動(dòng)眼神經(jīng)支配的眼外肌麻痹表現(xiàn)，患 眼呈不同程度的上瞼下垂和外斜視， 圖7 右側(cè)先天性動(dòng)眼神經(jīng)麻痹， 受累眼外斜視、正常的瞳孔集合反應(yīng)、輕微的上瞼下垂、上抬受限、 內(nèi)陷和內(nèi)收伴有輕度下斜和內(nèi)旋斜視。眼球向上、向下和向內(nèi)運(yùn)動(dòng)均 受限。眼球輕度突出。眼球向下轉(zhuǎn)時(shí)有內(nèi)旋轉(zhuǎn)。瞳孔改變 (常見)：瞳孔散大，對(duì)光反射和近反射消失，調(diào)節(jié)麻痹弱視 (常見)：由于視覺刺激減少，視力下降且不能矯正，先天性常見 假性Graefe 征 (不常見)：患眼試圖向下轉(zhuǎn)時(shí)，上瞼上舉假性Argyll-Robertson 瞳孔 (不常見)：患眼瞳孔散大，對(duì)光反應(yīng)消 失。當(dāng)眼球集合和向內(nèi)轉(zhuǎn)時(shí)，瞳孔收縮。水平注視眼瞼運(yùn)動(dòng)障礙 (不常見)：外轉(zhuǎn)時(shí)瞼裂變小，內(nèi)轉(zhuǎn)時(shí)瞼裂變寬動(dòng)眼神經(jīng)錯(cuò)向綜合征 (不常見)： 垂直方向注視時(shí)眼球后退、垂直方 向注視時(shí)眼球內(nèi)轉(zhuǎn)。
2, intracranial space occupying lesions (intracranialspaceoccupyinglesion): intracranial pressure increased intracranial pressure in the brain and endometrial tumors, generally all have said that the tentorial herniation has occurred. The performance of the patient's pupil dilation and photoreaction disappeared, the contralateral limb paralysis can occur, followed by contralateral pupil also appear to expand, accompanied by disturbance of consciousness. According to medical history and head CT can be more clear diagnosis. 3, cavernous sinus thrombosis and sinus aneurysm: can be expressed as cavernous sinus syndrome, in addition to oculomotor nerve paralysis, there are the first trigeminal nerve damage, orbital soft tissue, upper and lower eyelids, conjunctival, frontal scalp and Nasal congestion and edema, prominent or papilledema, inflammation often accompanied by symptoms of systemic infection, combined with orbital X-ray and lumbar puncture and blood tests can confirm the diagnosis. 4, supraorbital fissure syndrome and orbital apex syndrome: the former has a moving eye, pulley, abducens nerve and the first trigeminal dysfunction, the latter in addition to three cranial nerve damage, often accompanied by visual impairment, combined with Orbital optic nerve hole X-ray, blood tests, orbital CT and other more able to confirm the diagnosis. 5, meningitis: meningitis caused by oculomotor damage are mostly bilateral, and more with the tackle, abducens nerve involvement. Check the number of cerebrospinal fluid cells, protein quantitative bacteria. Key diagnoses Bacterial ptosis and strabismus (common): According to the oculomotor innervation of the extraocular muscle paralysis, the affected eyes were varying degrees of ptosis and exotropia, Figure 7 right side of the congenital oculomotor nerve paralysis, involvement Extrathoracic, normal pupil set reaction, slight ptosis, limited elevation, retraction and adduction accompanied by mild hypopynthus and pronation. Eye up, down and inward movement are limited. Eyeballs are slightly prominent. Eye turn down when there is rotation. Pupil changes (common): dilated pupils, loss of light reflex and reflex, paralysis of the amblyopia (common): reduced visual acuity due to decreased vision and can not be corrected, congenital common false Graefe sign (uncommon): affected eyes Pseudo Argyll-Robertson pupil (uncommon) on the upper eyelid when trying to turn down: the affected pupil becomes dizzy and reaction to light disappears. The pupil contracts as the eyeballs gather and turn inward. Horizontal fixation of eyelid dyskinesia (uncommon): external transitional fissure is small, internal transitional fissure widened oculomotor syndrome (not common): the vertical direction of the retroreflective gaze, the vertical direction of the eye when the turn.