嗜吞噬細胞無形體IgM免疫熒光試劑盒

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【公司名稱】 廣州健侖生物科技有限公司
【】    楊永漢 
【】 
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【公司地址】 廣州清華科技園創(chuàng)新基地番禺石樓鎮(zhèn)創(chuàng)啟路63號二期2幢101-3室
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加州舊金山分校的研究人員發(fā)現(xiàn)，每天喝碳酸飲料的人的白血細胞有更短的端粒。報告發(fā)表在《American Journal of Public health》期刊上。
端粒是染色體的末端區(qū)域，其長度與細胞衰老相關，更短的端粒被認為與慢性衰老疾病如心臟病、糖尿病和某些癌癥有關聯(lián)。
研究人員調查了年齡在20到65歲的5309位成年人飲用含糖飲料、非碳酸類飲料等的影響，發(fā)現(xiàn)每天消費20盎司碳酸飲料的人會增加4.6年的生理年齡，其對端粒長度的影響堪比吸煙。
中科院上海生科院營養(yǎng)科學研究所周斌研究組發(fā)現(xiàn)，在發(fā)育過程中心外膜祖細胞可轉變成心臟周圍的脂肪細胞，這一過程在成體穩(wěn)態(tài)心臟中并不發(fā)生，卻在心梗的模型中被再度激活。相關成果日前在線發(fā)表于《細胞研究》雜志。
心外膜祖細胞是一群具有分化多能性、位于心臟zui外層的細胞，研究發(fā)現(xiàn)其可分化成纖維細胞、血管平滑肌細胞甚至心肌細胞，但其轉化成脂肪細胞的能力鮮有報道。研究探索心外膜脂肪的起源，可為心臟冠狀動脈疾病的診斷和治療提供新的研究方向和思路。

Researchers at the University of California, San Francisco, found that white blood cells in people who drink carbonated drinks daily have shorter omeres. The report was published in the American Journal of Public health journal.
omere is the terminal region of the chromosome and its length is related to cellular senescence. Shorter omeres are thought to be associated with chronic aging diseases such as heart disease, diabetes and certain cancers.
Researchers investigated the effects of drinking 5,920 adults aged 20 to 65 years with sugary drinks, non-carbonated drinks and the like and found that those who consumed 20 ounces of carbonated soft drinks a year increased their physiological age by 4.6 years and had no effect on omere length The impact is comparable to smoking.
Chinese Academy of Sciences, Shanghai Institutes for Biological Sciences, Zhou Bin research group found that during development, epicardial progenitor cells can be transformed into cardiac fat cells around the heart, the process does not occur in adult steady heart, but in myocardial infarction model Was activated again. Related results recently published online in "Cell Research" magazine.
Epicardial progenitor cells, a group of differentiated pluripotent cells located in the outermost layers of the heart, have been found to differentiate into fibroblasts, vascular smooth muscle cells and even cardiomyocytes, but their ability to transform into adipocytes has rarely been reported. Studying the origin of epicardial fat can provide new research directions and ideas for the diagnosis and treatment of coronary heart disease in the heart.